Tuesday, January 30, 2018

Alarming new study: Alzheimer’s disease may spread through blood transfusions

Alzheimers disease may spread through blood transfusions

Can you catch Alzheimer’s? Can Alzheimer’s disease spread through blood transfusion? These questions have been asked by many, but there were no positive proof. But now, a new study suggests the disease might spread through blood transfusion.

Researchers from the University of British Columbia have found that healthy mouse who shared blood with another mouse who had Alzheimer's plaques did actually develop beta-amyloid plaques in its brain.

However, Professor Weihong Song, the lead researcher of the study said, people shouldn’t be frightened about catching Alzheimer’s disease through blood transfusions.


He said that in humans, beta-amyloid protein may pass through blood transfusions whether they have Alzheimer’s or not, because the protein can be manufactured outside the brain.

However, the chances of a healthy person developing beta-amyloid plaques in the brain via blood transfusion are very little because very low level of beta-amyloid can be exchanged. 

Beta-amyloid plaques are one of the hallmarks of Alzheimer’s disease. They are found between neurons in the brain. Beta-amyloid is a protein bit clipped from an APP (amyloid precursor protein). Inside the brain of a healthy person, these protein fragments get broken down and removed. However, in Alzheimer's patients, these sticky fragments gather to form solid, insoluble senile plaques.


Beta-amyloid protein starts to kill synapses – connections between neurons. After destroying synapses the protein clumps into senile plaque, which destroys the nerve cells.

The death of nerve cells cause the Alzheimer’s patient to gradually loose memory, learning and the person eventually becomes unfit to perform simple everyday tasks like tying shoelaces.

Alzheimers disease may spread through blood transfusions
Alzheimer's cells showing beta-amyloid plaques
For their study, the team inserted beta-amyloid gene into healthy mice, which enabled the rodents to develop beta-amyloid plaques similar to the type seen in human Alzheimer’s patients.

The researchers then surgically attached each beta-amyloid gene carrying mouse to a mouse without the gene. The process simulated a shared blood system.

The healthy mice started to amass beta-amyloid in their brains, and after remaining attached for a year, the healthy mice developed Alzheimer’s. In the areas of the brain that were crucial for learning and memory, the infected mice showed destructive activities observed in Alzheimer’s.

Prof. Song said the protein can enter the brain from an attached mouse with beta-amyloid and cause neurodegeneration. [মধুর যত মধুর গুণাবলী]

A different study conducted last year, showed approximately 1.4 million who received blood transfusions from people with Parkinson’s disease and other forms of dementia in the UK, weren’t likely to get the diseases.

Professor Tara Spires-Jones of the University of Edinburgh, in the UK, said this is vital for us to understand the biological changes in our body and how toxic proteins spread, but this is very distant from Alzheimer’s disease in humans.

3 comments:


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